Drug-Induced Acute Tubulointerstitial Nephritis
نویسندگان
چکیده
منابع مشابه
Inadequate drug prescription and the rise in drug-induced acute tubulointerstitial nephritis incidence
Drugs are a frequent cause of acute tubulointerstitial nephritis (ATIN). Antibiotics, non-steroidal anti-inflammatory drugs and recently proton pump inhibitors stand among the most commonly responsible ones. However, their respective responsibility is not well known. This study reports 33 cases of drug-induced ATIN (DI-ATIN), the most frequent ones being metamizole and omeprazole. Clinicians of...
متن کاملAcute tubulointerstitial nephritis/drug induced acute kidney injury; an experience from a single center in Pakistan.
INTRODUCTION There is no information in literature specifically on the prevalence and clinicopathological characteristics of acute tubulointerstitial nephritis/drug induced acute kidney injury (AKI) from Pakistan. OBJECTIVES We aim to report a series of cases from patients developing AKI after exposure to some medications or finding of interstitial nephritis on histopathology. PATIENTS AND ...
متن کاملAcute and Chronic Tubulointerstitial Nephritis
Cellular and fluid exudation in the interstitial tissue was noted by Councilman in 1898, while he studied kidneys of patients who died of scarlet fever and diphtheria (1). Councilman also determined that these kidneys did not contain bacteria (they were sterile). He called the condition acute interstitial nephritis (AIN). The term interstitial nephritis connotes predominant involvement of the r...
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BACKGROUND AND AIMS Acute tubulointerstitial nephritis (ATIN) is a potentially reversible cause of acute kidney injury with the majority of cases drug related. Our aims were to examine the incidence profile of patients with ATIN in Scotland and to assess the impact of corticosteroid treatment. DESIGN AND METHODS All adult patients with biopsy-proven ATIN, diagnosed between 2000 and 2012, pres...
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Four days later (after 3 weeks of complete anuria) diuresis started to resume. Urinalysis revealed proteinuria (4.12 g/24 h), lambda light chains (2.41 g/l) and microhaematuria. Subsequently, complement normalized and urinalysis became negative. Creatinine dropped to 106mmol/l and no paraprotein was detectable in neither serum nor urine. In conclusion, this report describes one of the oldest pa...
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ژورنال
عنوان ژورنال: Internal Medicine
سال: 2020
ISSN: 1220-5818
DOI: 10.2478/inmed-2020-0127